Defective skeletal muscle growth in lamin A/C-deficient mice is rescued by loss of lap2α.

Authors

Tatiana V. Cohen, Children's National Medical Center, Washington, DCFollow
Viola F. Gnocchi, Children's National Medical Center, Washington, DC
Jonathan E. Cohen, Children's National Medical Center, Washington, DC
Aditi Phadke, George Washington University
Henry Liu, Children's National Medical Center, Washington, DC
Juliet A. Ellis, King's College London
Roland Foisner, Medical University, Vienna, Austria
Colin Stewart, Institute of Medical Biology, Immunos, Singapore
Peter S. Zammit, King's College London
Terence A. Partridge, Children's National Medical CenterFollow

Document Type

Journal Article

Publication Date

7-2013

Journal

Human Molecular Genetics

Volume

Volume 22, Issue 14

Inclusive Pages

2852-2869

Keywords

DNA-Binding Proteins--deficiency; Lamin Type A--deficiency; Membrane Proteins--deficiency; Muscular Dystrpophy, Emery-Dreifuss--metabolism

Comments

PMCID: PMC3888145 [Available on 2014/7/15]

APA Citation

Cohen, T. V., Gnocchi, V. F., Cohen, J. E., Aditi, P., Liu, H., Ellis, J. A., . . . Partridge, T. A. (2013). Defective skeletal muscle growth in lamin A/C-deficient mice is rescued by loss of lap2α. Human Molecular Genetics, 22(14), 2852-2869.

Peer Reviewed

1