T cells lacking HDAC11 have increased effector functions and mediate enhanced alloreactivity in a murine model.
Document Type
Journal Article
Publication Date
7-13-2017
Journal
Blood
Volume
130
Issue
2
Inclusive Pages
146-155
DOI
10.1182/blood-2016-08-731505
Keywords
Adoptive Transfer; Animals; B-Lymphocytes; Chromatin; Chromatin Immunoprecipitation; Disease Models, Animal; Gene Expression Regulation, Neoplastic; Graft vs Host Disease; Histone Deacetylase 1; Interferon-gamma; Lymphocyte Activation; Lymphoma, B-Cell; Mice; Mice, Knockout; Promoter Regions, Genetic; Signal Transduction; T-Box Domain Proteins; T-Lymphocytes; Tumor Necrosis Factor-alpha
APA Citation
Woods, D., Woan, K., Cheng, F., Sodré, A., Wang, D., Wu, Y., Wang, Z., Chen, J., Powers, J., Pinilla-Ibarz, J., Yu, Y., Zhang, Y., Wu, X., Zheng, X., Weber, J., Hancock, W., Seto, E., Villagra, A., Yu, X., & Sotomayor, E. M. (2017). T cells lacking HDAC11 have increased effector functions and mediate enhanced alloreactivity in a murine model.. Blood, 130 (2). http://dx.doi.org/10.1182/blood-2016-08-731505
Peer Reviewed
1
