Loss of Hypermethylated in Cancer 1 (HIC1) in breast cancer cells contributes to stress-induced migration and invasion through ?-2 adrenergic receptor (ADRB2) misregulation.

Authors

Gaylor Boulay, Harvard University
Nicolas Malaquin, Université de Lille 1
Ingrid Loison, Université de Lille 1
Benedicte Foveau, Jewish General Hospital,
Capucine Van Rechem, Jewish General Hospital,
Brian R. Rood, George Washington UniversityFollow
Albin Pourtier, Université de Lille 1
Dominique Leprince, Université de Lille 1

Document Type

Journal Article

Publication Date

2-16-2012

Journal

Journal of Biological Chemistry

Volume

Volume 287, Issue 8

Inclusive Pages

5379-5389

Keywords

Breast Neoplasms--pathology; Cell Movement--genetics; Kruppel-Like Transcription Factors--metabolism; Receptors; Adrenergic; beta-2--genetics; Stress; Physiological--genetics

Comments

This is an open access PubMed Central article. Click on link for full-text access.

APA Citation

Boulay, G., Malaquin, N., Loison, I., Foveau, B., Van Rechem, C., Rood, B. R., . . . Leprince, D. (2012). Loss of hypermethylated in cancer 1 (HIC1) in breast cancer cells contributes to stress-induced migration and invasion through β-2 adrenergic receptor (ADRB2) misregulation. Journal of Biological Chemistry, 287(8), 5379-5389.

Peer Reviewed

1

Open Access

1