Tissue Acylcarnitine Status in a Mouse Model of Mitochondrial β-oxidation Deficiency during Metabolic Decompensation due to Influenza Virus Infection.

Authors

Tatiana N Tarasenko
Kristina Cusmano-Ozog, George Washington University
Peter J McGuire

Document Type

Journal Article

Publication Date

9-1-2018

Journal

Molecular genetics and metabolism

Volume

125

Issue

1-2

Inclusive Pages

144-152

DOI

10.1016/j.ymgme.2018.06.012

Keywords

Acyl-CoA Dehydrogenase, Long-Chain; Animals; Cardiomyopathies; Carnitine; Disease Models, Animal; Fatty Acids; Female; Humans; Hypoglycemia; Lipid Metabolism, Inborn Errors; Lipid Peroxidation; Liver; Liver Failure; Metabolic Diseases; Mice; Mitochondrial Diseases; Muscle, Skeletal; Muscular Diseases; Myocardium; Oxidation-Reduction

APA Citation

Tarasenko, T., Cusmano-Ozog, K., & McGuire, P. (2018). Tissue Acylcarnitine Status in a Mouse Model of Mitochondrial β-oxidation Deficiency during Metabolic Decompensation due to Influenza Virus Infection.. Molecular genetics and metabolism, 125 (1-2). http://dx.doi.org/10.1016/j.ymgme.2018.06.012

Peer Reviewed

1