The cancer-associated FGFR4-G388R polymorphism enhances pancreatic insulin secretion and modified the risk of diabetes

Authors

Shereen Ezzat, Ontario Cancer Institute, Toronto, ON
Lei Zheng, Ontario Cancer Institute, Toronto, ON
Jose C. Florez, Harvard University
Norbert Stefan, University of Tübingen, Germany
Thomas Mayr, Max Planck Institute of Biochemistry, Martinsried, Germany
Maw Maw Hliang, Ontario Cancer Institute, Toronto, ON
Kathleen A. Jablonski, George Washington UniversityFollow
Maegan Harden, Broad Institute of MIT and Harvard, Cambridge, MA
Alena Stancakova, University of Eastern Finland
Markku Laakso, University of Eastern Finland

Document Type

Journal Article

Publication Date

2013

Journal

Cell Metabolism

Volume

Volume 17, Issue 6

Inclusive Pages

929-940

APA Citation

Ezzat, S., Zheng, L., Florez, J.C., Stefan, N., Mayr, T., Hliang, M.M., Jablonski, K., Harden, M., Stančáková, A., Laakso, M. (2013). The cancer-associated FGFR4-G388R polymorphism enhances pancreatic insulin secretion and modifies the risk of diabetes. Cell Metabolism, 17(6), 929-940.

Peer Reviewed

1